123). It really is notable that GLS2 is tumorigenic in breast cancer124, and its expression could compensate for loss of Gls in MYC-induced mouse liver tumours40. This implies that GLS2 could confer resistance to GLS1-distinct inhibitors, such as the allosteric inhibitor BPTES or CB-839, and this resistance may very well be hypothetically avoided b
The NRX-1532 Diaries
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